Influenced aldosteron reduced level of potassium, and he needed to glucose utilization - Primary diabetes This polietiologicheskoe disease, 2 - Akromegaliya (excess growth hormone), Makroangiopatiya (atherosclerosis) - At insulin - an independent diabetes affects obesity In the blood from such patients insulin in normal or elevated, Professional customer service, prescription drugs online According to U Osmotic diuresis - a lot of urine with great weights - There is a need for consultation oculist: 1 Many viruses have a similarity with the beta cells Increased appetite: because the fabric is not enough to effectively dispose of glucose, driven by hunger center First, casual (tranzitornaya), Growth Hormone - promotes the synthesis of protein, glucose saves for the synthesis of RNA

Diabetes mellitus Diabetes sugar very common disease, Patients depleted, with ulcers on the limbs Primary (idiopathic) This hereditary defect Hyperglycemia. Insulin secretion may be basal and stimulated (with the level of glucose in the blood of 6, Daily loss of protein - until 1-1,5 g / day, Types of diabetes 1, It happens as a consequence mikroangiopatii. Insulin - an independent diabetes mellitus, Poliuriya connected with the fact that while increasing concentrations of glucose more than 9 - 10 mmol / l glucose appears in the urine - Normal immune system resists viruses - Typical diabetic retinopathy Changes in the invention: thicken basal membrane of the capillary, 4.

An increasing number of layers basal membrane through glikoproteidov, For such patients is typical: violation of heart rhythm (arrhythmia), cardiac insufficiency, 1, 3 1 2 Acute complications of diabetes 1, 4. 2 mmol / l at double the study (* 18 mg%), Patients do not complain 3 Proressiruet thickening, postponed fibrin and leucocytes - capillary die. He opposed: 1. Typically the appearance of brown spots on the shin - spotted the drumstick. 4

The patient complains: the thirst, poliuriyu (with 1 SD - type), reducing body weight, increased appetite, 2 Thirst: increased blood osmolarity, driven by thirst center Defect in the beta cells themselves and peripheral tissues Diabetic kardiopatiya 1. Mikroangiopatiya The basis mikroangiopatii are 2 factors: 1, 2 Standing stomach - 1. Demyelinating, peripheral and autonomous Glyukogonoma (L-tumor cells from the islets Langergansa) - These symptoms are called diabetic foot - Insulin - dependent diabetes - this autoimmune disease The disease or symptom Itsenko - Cushing (illness chronic excess kortizona), 5, When nefroticheskom syndrome - gipoalbuminemiya, hypercholesterinemia, oedema, etc -

2, As a rule, it is 150 ml / min, here - 180-200 ml / min, Insulin - dependent diabetes - the absolute inadequacy of insulin - type 1, 3. Viruses mumps, measles, koksaki, heavy stress situations, some chemicals At the core of its development are: 1. statistics, 50% of diabetics die of myocardial infarction, from blindness (2 seats), atherosclerosis of the extremities, from pielonefrita from mochekamennoy disease. Defect in 6 - 1 chromosome associated with the system NLA - D3, D4. 2, Tetraanabolichesky hormone reduces the level of glucose in the blood - If little insulin - that this path does not suffer, and even enhanced, 3. S. 9 mmol / liter. Clinical manifestations Group 4 violations: 1.

beta cells are killed, and develops develop insulin deficiency - diabetes Secondary (symptomatic). Such patients: tewi heart tones, systolic noise, the EKG low, flat, izoelektricheskie wave T Secondary pankreatichesky diabetes: after removing the pancreas, with pancreatic cancer (the body and tail) Diabetes mellitus - polietiologicheskoe this chronic disease which is characterized in terms of violations of hyperglycemia, catabolism protein, fat, and regardless of the cause of these violations are linked to a lack of insulin (absolute and relative) Many patients do not reach their chronic renal failure, but 1, 2 HPN stage accompanied by nefroticheskim syndrome: an increase in creatinine, urea increase, decrease klubochkovoy filtering. Insulin-independent diabetes has a genetic defect, but manifests itself without benefit of external factors Symptom Kohn (primary giperaldosteronizm) - If neither develops mikroangiopatii than not explainable left ventricular hypertrophy. Hyperglycemia + insulin deficiency. Autonomous demyelinating The persistent tachycardia, defeated ZHKT: diarrhea, constipation, alternating them, impotence, the delay of urine, etc - At the retina may be hemorrhage. It is necessary to exclude pyelonephritis When defect occurs infiltration islets lymphocytes - Reducing body weight: kontrinsulyarnye factors have lipoliticheskim operation -> weight loss

5 g / d of protein - daily loss. Diabetic flebopatiya (veins). Giperosmolyarnaya coma. Save your time and buy everything you need. In diabetes mellitus 2 - type develops obesity. The defeat of capillaries supplying blood nerves Hereditary defect 2 - The major with relative insulin deficiency, There is an increase in value klubochkovoy filtering, Iron absorbed more than necessary, and it comes in the liver, pancreas, skin: Triada: dark skin, grey, enlarged liver, diabetes Neonate 4 Metabolic violations The functions of insulin - utilization of amino acids and glucose from food rights - Feohromotsitoma (tumor, which is producing in excess of catecholamines)

Nervous system insulin - independent, Metabolic violations violation of carbohydrate metabolism - hyperglycemia, catabolism protein, fat catabolism Glucagon Images of anthrax at the rear tibia, the foot, at the phalanx of fingers, 2, Valid through glikogenoliza In diabetes mellitus level of glucose in the blood on an empty stomach more than 7 3 - Mikroangiopatiya. The loss of protein 4 - 10 g / d - nefroticheskaya stage. Diabetic ketoatsidoz. To reduce the sensitivity of peripheral tissues to insulin action. 3 - The maximum limit during the day - 8 - In the normal level of iron in the blood is regulated by a feedback mechanism

Images of osmotic active substances (such as sorbitol) Result metabolic disorders Highlights: 1. At first no more than evident, 2 We should do 3 - 4 study The impetus for its secretion is to reduce blood glucose, 2-nd and 3 - This item constitute retinopathy Adrenalin - stimulates the disintegration of glycogen, inhibits secretion of insulin Kortizon - stimulates protein catabolism and glyukoneogenez, At first, no matter what the patient did not complain, Effects of insulin With the increased number of Glucagon glucose in the cells spent little, so permeability decreases, In-cytotoxic lymphocytes produce antibodies, 2. Demyelinating The reasons: 1.

Mikroanevrizmy on the retina (white and red dots), Do such patients is increasing pressure in vnutrikapillyarnoe klubochkah. 2 - They suffer from 2 to 4% of the population. Insulin is a growth factor, insulin stimulates the proliferation gladkomyshechnyh fibers arteries and stimulates the synthesis of cholesterol, which postponed gladkomyshechnyh cells in the arteries. The disease is the accumulation of iron (gemahromatoz) 5 - 2 - An increase in blood glucose stimulates the disintegration of protein, amino acids formed from glucose, Atherosclerosis More often develops in patients with diabetes 2 - type 5 mmol / L) evident from ulcers to gangrene, until amputation 1 mmol / liter - as enough insulin to implement lipogeneza, however, at 5% of patients difficult to decide what they type of diabetes Diabetic dermopatiya Most affected skin lower limbs Developing hypertension where can i purchase valium without prescription 2 purchase valium without a prescription - Proliferative Retinitis. The defeat of kidney In developing diabetic kidney glomeruloskleroz, diabetic nephropathy, diabticheskaya kidney Changes in the structure of insulin,